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  • Home
  • APOE U19
    • What is APOE?
    • Research Program
    • Publications
  • Data
    • Gene Expresssion Database
    • Lipid Abundance Database
    • Bulk RNA-seq Database
    • Metabolomics Database
    • APOE U19 Publications Database
    • Human CSF Immune Activation Markers
    • Other
    • Brain Lysate Data
      • Soluble Fraction: TBS
      • Detergent Soluble Fraction: TBS-X
      • Insoluble Fraction: Neutralized FA
  • Resources
    • Antibodies
    • Animal Models
    • IPSC Models
  • Protocols
    • iPSC Protocols
    • Published Methods
  • About us

ApoE and Other Neurodegenerative Disorders

Neurodegenerative disease concomitant proteinopathies are prevalent, age-related and APOE4-associated. Robinson, J. L., Lee, E. B., Xie, S. X., Rennert, L., Suh, E., Bredenberg, C., Caswell, C., Van Deerlin, V. M., Yan, N., Yousef, A., Hurtig, H. I., Siderowf, A., Grossman, M., McMillan, C. T., Miller, B., Duda, J. E., Irwin, D. J., Wolk, D., Elman, L., McCluskey, L., Trojanowski, J. Q. (2018).  Brain : a journal of neurology, 141(7), 21812193.

Gene-environment interaction between lead and Apolipoprotein E4 causes cognitive behavior deficits in mice. Engstrom AK, Snyder JM, Maeda N, Xia Z.  Mol Neurodegener. 2017 Feb 7;12(1):14.

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